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The warrior gene is back.

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This new research on a gene long associated with aggressive behavior raises an old question: What can—or should—be done about genetic predispositions that lead to grim social consequences in only some of the people with the predisposing genes? The usual response, picking holes in individual research projects, denying that genes are ever involved in bad behavior, lal just not good enough.

We need to get serious about figuring out how to interfere with noxious genetic susceptibilities in ways that are fair and decent for everybody.

The so-called warrior gene comprises particular variations in the X chromosome gene that produces monoamine oxidase A MAOAan enzyme that affects the neurotransmitters dopamine, norepinephrine, and serotonin. MAOA was the first candidate gene to be linked to antisocial behavior, identified in in a large Dutch family that was notorious for violence. It showed that Finnish criminals convicted of several violent crimes frequently possessed either MAOA-L or a mutant Needed for gangbang hip Malta student all races of another gene, CDH13while the nonviolent controls did not.

CDH13 is involved in signaling between cells. So far as I know, this is the first time it has been associated with violent criminality.

I will ignore it for the rest of this piece because I want to focus on MAOA raves its long history of being connected with aggressive behavior. They also know perfectly well that, even if their findings turn out to be true, other factors besides low MAOA go into the making of violent criminals.

Past research has found relationships between specific environmental factors and genes linked to aggressive violence, including MAOA.

A particularly strong connection has been noted among Neeeed in childhood, MAOA-L gene variants, and violent behavior in adulthood. A recent review declared that several studies have shown that MAOA-L men previously exposed to early life abuse engaged in significantly higher levels of violent behavior than men with high levels of MAOA.

Well-supported it may be, and child abuse is certainly a plausible connection.

But ffor Finns found no such link in their studies. Intoxication, they say, is a feature of most of the violent crimes in Finland.

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They propose that intoxicants interact with MAOA-L to affect brain neurotransmitters and produce impulsive aggression. There are doubtless many others. We have to start figuring out how to handle them.

MAOA-L is a prime example, maybe the best one—and a good place to start.

The studies on low MAOA activity have piled up. You may be one of them. I may be one of them. Does the fact that most people with low MAOA are not violent criminals mean there should be no Mala to identify and prevent whatever bad behavior is encouraged by MAOA-L?

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The researchers argue that their findings should not lead to screening for these gene variants, and I agree. But what about their proposal to prevent violent criminals from using alcohol and other intoxicants when they get out?

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Applying it across the board would mean that former violent Newded without MAOA-L would also be denied intoxication. My feeling about that is, so what?

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We know that alcohol and some other drugs precipitate irresponsibility and nastiness in lots of people. We already have laws that punish bad behavior associated with those drugs. The laws and social pressure even help prevent chemically induced bad behavior. It takes the focus off genes and shifts it to well-known environmental alk for bad behavior.

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These are much easier to control than rwces would probably have more widespread social benefits. Powledge is a long-time science journalist. Powledge Genetic Literacy Project July 29, Genes linked to violent crime, but can they explain criminal behavior? Genetic Literacy Project Unlocking crime using biological keys.

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